Upregulation of inflammatory genes and downregulation of sclerostin are key elements for fracture healing

Journal of Translational Medicine

Poster Presentation

T Monjardino¹  R Lucas¹  J Caetano-Lopes²  I P Perpétuo²  A Lopes²  D Fernandes²  H Canhão³  A Rodrigues³  J Monteiro⁴  Y T Konttinen⁵ 

[1] Dept. of Hygiene and Epidemiology, University of Porto Medical School, Porto, Portugal;Institute of Public Health, University of Porto, Porto, Portugal;Rheumatology Research Unit, Instituto de Medicina Molecular, Faculdade de Medicina da Universidade de Lisboa, Lisbon, Portugal;Rheumatology Research Unit, Instituto de Medicina Molecular, Faculdade de Medicina da Universidade de Lisboa, Lisbon, Portugal;Serviço de Reumatologia e Doenças Ósseas Metabólicas, Hospital de Santa Maria, Lisbon, Portugal;Serviço de Ortopedia, Hospital de Santa Maria, Lisbon, Portugal;University of Helsinki, Dept. of Medicine; ORTON Orthopaedic Hospital of the Invalid Foundation, Helsinki, COXA Hospital for Joint Replacement, Tampere, Finland;

关键词: Trabecular Bone;    Bone Remodel;    Fracture Healing;    Callus Formation;    Biomechanical Property;   

DOI  :  10.1186/1479-5876-8-S1-P68

来源: Springer

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© Fonseca et al; licensee BioMed Central Ltd. 2010. This article is published under license to BioMed Central Ltd.