International Journal of Molecular Sciences | |
Loss of p16Ink4a Function Rescues Cellular Senescence Induced by Telomere Dysfunction | |
Xiufeng Zhang1  Xiaoming Wu1  Wenru Tang1  | |
[1] Faculty of Environmental Science and Engineering, Kunming University of Science & Technology, Kunming 650224, China; E-Mails: | |
关键词: p16Ink4a; Werner Syndrome; telomere dysfunction; | |
DOI : 10.3390/ijms13055866 | |
来源: mdpi | |
【 摘 要 】
p16Ink4a is a tumor suppressor and a marker for cellular senescence. Previous studies have shown that p16Ink4a plays an important role in the response to DNA damage signals caused by telomere dysfunction. In this study, we crossed
【 授权许可】
CC BY
© 2012 by the authors; licensee Molecular Diversity Preservation International, Basel, Switzerland.
【 预 览 】
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