Skeletal muscle mTORC1 regulates neuromuscular junction stability

Journal of Cachexia, Sarcopenia and Muscle

Simona Boncompagni¹  Aram Megighian²  Marco Pirazzini²  Hendrik Nolte³  Clara Türk³  Marcus Kruger⁴  Francesca Solagna⁵  Martina Baraldo⁵  Leonardo Nogara⁵  Alessia Geremia⁵  Vanina Romanello⁵  Bert Blaauw⁵  Marco Sandri⁵ 

[1] CeSI‐Met—Center for Research on Ageing and Translational Medicine and DNICS, Department of Neuroscience, Imaging and Clinical Sciences, University G. d' Annunzio, Chieti, Italy;Department of Biomedical Sciences, University of Padova, Padova, Italy;Institute for Genetics, Cologne Excellence Cluster on Cellular Stress Responses in Aging‐Associated Diseases (CECAD), Cologne, Germany;Institute for Genetics, Cologne Excellence Cluster on Cellular Stress Responses in Aging‐Associated Diseases (CECAD), Cologne, Germany;Center for Molecular Medicine (CMMC), University of Cologne, Cologne, Germany;Venetian Institute of Molecular Medicine (VIMM), Padova, Italy;Department of Biomedical Sciences, University of Padova, Padova, Italy;

关键词: mTOR;    NMJ;    Autophagy;    Mitochondrial dysfunction;   

DOI  :  10.1002/jcsm.12496

来源: Wiley

【 授权许可】

CC BY