ON the pathogenesis of headache following TIA

Arquivos de Neuro-Psiquiatria

Charles André¹  Maurice B. Vincent² 

[1] ,Universidade Federal do Rio de Janeiro Hospital Universitário Clementino ,Brasil

关键词: transient cerebral ischemia;    Cortisol;    headache;    neuroendocrine system;    serotonin;    trigeminovascular system;    cefaléia;    cortisol;    isquemia cerebral transitória;    serotonina;    sistema neuroendócrino;    sistema trigeminovascular;   

DOI  :  10.1590/S0004-282X1997000200001

来源: SciELO

【 摘 要 】

Twelve out of 49 patients with single or multiple transient ischemic attacks (TIAs) had TIA-related headaches, mostly in close temporal relation to the ischemic onset. Headache predominated in patients taking vasodilators when TIA occurred or with orthostatic hypotension at the first clinical examination, but arterial hypertension or a personal history of migraine were not more frequent in patients with headache. The site of the pain did not correlate with the presumed territory of cerebral ischemia. Pain during TIA is conceivably due to an interaction between cerebral vessels and the surrounding nervous system. Blood vessels have a sturdy physiological role corcerning blood flow regulation, with receptors and signaling molecules potentialy involved with pain production. Reflex mechanisms should justify pain in other areas.

【 授权许可】

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