The TWEAK/Fn14 pathway as an aggravating and perpetuating factor in inflammatory diseases; focus on inflammatory bowel diseases

Journal of Leukocyte Biology

Linda C. Burkly²  Taeko Dohi²  Linda C. Burkly ³  *²  Taeko Dohi and¹ 

[1] Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan;;Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan; and Department of Immunology, Biogen Idec, Cambridge, Massachusetts, USA Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan; and Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan; and Department of Immunology, Biogen Idec, Cambridge, Massachusetts, USA Department of Immunology, Biogen Idec, Cambridge, Massachusetts, USA Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan; and Department of Immunology, Biogen Idec, Cambridge, Massachusetts, USA Department of Immunology, Biogen Idec, Cambridge, Massachusetts, USA Department of Gastroenterology, Research Center for Hepatitis and Immunology, Research Institute, National Center for Global Health and Medicine, Tokyo, Japan;

关键词: Crohn's disease;    ulcerative colitis;    TNF superfamily;    epithelial death;    inflammation;    tissue remodeling;   

DOI  :  10.1189/jlb.0112042

学科分类：生理学

来源: Federation of American Societies for Experimental Biology

【 授权许可】

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